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Exendin-4 improves resistance to Listeria monocytogenes infection in diabetic db/db mice.

J Vet Sci · 2012

Last updated 2026-05-28

In a study on diabetic mice, treatment with exendin-4—a GLP-1 drug—improved their ability to fight infection from Listeria bacteria. The drug reduced obesity and high blood sugar, enhanced the function of immune cells called macrophages, and led to milder liver damage during infection compared to untreated mice.

AI summary of the abstract below.

JournalJ Vet Sci, 2012
Citations3
Relative citation ratio0.10
NIH percentile7
Molecules
Conditions studied Type 2 Diabetes

Abstract

The incidence of diabetes mellitus is increasing among companion animals. This disease has similar characteristics in both humans and animals. Diabetes is frequently identified as an independent risk factor for infections associated with increased mortality. In the present study, homozygous diabetic (db/db) mice were infected with Listeria (L.) monocytogenes and then treated with the anti-diabetic drug exendin-4, a glucagon-like peptide 1 analogue. In aged db/db mice, decreased CD11b(+) macrophage populations with higher lipid content and lower phagocytic activity were observed. Exendin-4 lowered high lipid levels and enhanced phagocytosis in macrophages from db/db mice infected with L. monocytogenes. Exendin-4 also ameliorated obesity and hyperglycemia, and improved ex vivo bacteria clearance by macrophages in the animals. Liver histology examined during L. monocytogenes infection indicated that abscess formation was much milder in exendin-4-treated db/db mice than in the control animals. Moreover, mechanistic studies demonstrated that expression of ATP binding cassette transporter 1, a sterol transporter, was higher in macrophages isolated from the exendin-4-treated db/db mice. Overall, our results suggest that exendin-4 decreases the risk of infection in diabetic animals by modifying the interaction between intracellular lipids and phagocytic macrophages.

Verbatim abstract via PubMed 23000581 ↗